Epidemics of seasonal influenza occur due to influenza A or B viruses. Due to the segmented nature of the influenza genome, in which coding sequences are located on individual RNA strands ...

Influenza viruses are among the most likely triggers of future pandemics. A research team has developed a method that can be used to study the interaction of viruses with host cells in unprecedented ...

One genome segment was derived from a classical swine H1N1 lineage ... human, and classical swine influenza A viruses. Triple reassortant viruses are established in the US and Canada, but also have ...

or antigenic shift (which for flu is usually the acquisition of a whole new segment of RNA). Any scientist or physician interested in the influenza virus itself, and all its fascinating ...

A research team led by the School of Public Health in the LKS Faculty of Medicine, the University of Hong Kong (HKUMed), in ...

The influenza virus manipulates the body's gene regulation system to accelerate its own spread, according to researchers at ...

The virus’s genome includes segments derived from the Eurasian, classical, and “triple-reassortant” swine influenza lineages ... from Mexico reveals 18 that match the 2009 viral lineage ...

Scientists at the United States Department of Agriculture's National Animal Disease Center, with multiple academic, state and ...

Pigs are the most important reservoir for influenza A viruses, acting as an intermediary host for both interspecies transmission and genetic reassortment. This is due to the high susceptibility of ...

The influenza virus causes infection in the same way as most viruses: invading a body cell in your nose or throat, high-jacking the cell’s machinery, and using that to replicate its own genome.

Viruses that are enveloped include HIV, influenza, Ebola, dengue, and chikungunya. A field that attempts to describe the functions and interactions of genes and proteins by using genome-wide ...

More information: Lukas Broich et al, Single influenza A viruses induce nanoscale cellular reprogramming at the virus-cell interface, Nature Communications (2025). DOI: 10.1038/s41467-025-58935-8 ...